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35) 22 sin(α)sin(β) 2sinα β cosα β 22 cos(α)cos(β)2sinαβsinαβ 22 sin(α)sin(β) 2cosα β sinα β Formulae of differential calculus Product rule d (uv)udvvdu dx dxdx xystem Quotient rule Chain rule du vduudv dx dx dx v v2 d f(g(x))df×dg dx dg fx Derivatives of elementary functions d xn nxn1 dx d exp(ax)aexp(ax) dx d ln(x)1 dx x d sin(x)cos(x) dx d cos(x)sin(x) dx d sin1 x 1 dx 1x2 dtan1x 1 dx 1 x2 594 Failure detectors More precisely, which is able to explain the generation of the high frequency (HF), low frequency (LF), and very low frequency (VLF) peaks existing in the heart rate variability (HRV) spectrum.
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© 2005 by Taylor Francis Group, LLC hydronephrotic kidney model 100 isolated perfused rat kidney 91 radiocontrast agents 488 atriopeptin isolated renal microvessels 101 atropine isolated renal microvessels 100 auranofin 307, 311, 312 aurothiomalate (sodium) 308 autoantigens immune response 52 autocoids 280 autoimmune diseases acute cyclosporine nephrotox.
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See also M2 ion channel inhibitors Aminoglycosides, Vol 1, 8789, 171179, 278279; Vol 2, 1228 amikacin, Vol 1, 179, 278, 281 aminoglycoside acetyltransferases (AACs), Vol 1, 8889, 176178 aminoglycoside adenylyltransferases, Vol 1, 175176; Vol grading, 1228 AAC(3) family, Vol 1, 178 Gcn5-related N-acetyltransferases, Vol 1, 177 major subclasses, Vol 1, 177 aminoglycoside nucleotidyltransferases (ANTs), Vol 1, 89, 176; Vol 2, 1228 aminoglycoside phosphotransferases (APHs), Vol 1, 89, 176; Vol 2, 1228 armA gene, Vol 1, 179 bacterial cell uptake, Vol 1, 171 bacterial resistance, Vol 1, shstem cross resistance and alternative agents, Vol 1, 179, 281282 dibekacin, 179 efflux-mediated resistance, Vol 1, 174 enzymatic modification, Vol 1, 8789, 174178, 1228 mechanism of action, 171173 mechanisms of resistance, 8789, 173178 Mycobacterium tuberculosis, Vol 1, 278279 nuclear magnetic resonance (NMR) structure, Vol 1, 171172 pseudo-polysaccharides, Vol 1, 171 ribosomal ambiguity (ram) state, Vol 1, 172173 ribosomal mutations, Vol 1, 173 16s rRNA methylation, Vol 1, 173174 Theorell-Chance kinetic mechanism, Vol 1, 176 transposons, Vol 1, 178 uptake into cytoplasm, 171 Amoxicillin, Vol 2, 684 Amphotericin B Vol 1, 295303 mechanism of action, Vol 1, 297, 335, 580; Vol 2, 932, 987, 1107 interaction with sterols, Vol 1, 298299 oxidative damage, Vol 1, 299 polyene resistance emergence, Vol 1.
7 The following pictures represent silicate anions. Tumor suppressor p53 is a direct transcriptional activator of the human bax gene. Chem Phys Lipids sstting Lopez-Rodriguez ML, Viso A, Ortega-Gutierrez S, Lastres-Becker I, Gonzalez S, Fernandez- Ruiz JJ. 8) This implies that the flow residence time m ̇ is critical for the inhaled air to be heated, given that the heat source is driven by the difference in the constant wall temperature and the inhaled air.
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